Cannabinoids can prevent cancer, reduce heart attacks by 66% and insulin dependent diabetes by 58%. Cannabis clinician Dr. William Courtney recommends drinking 4 – 8 ounces of raw flower and leaf juice from any Hemp plant, 5 mg of Cannabidiol (CBD) per kg of body weight, a salad of Hemp seed sprouts and 50 mg of THC taken in 5 daily doses.
Cannabis – whether Sativa, Indica, Ruderalis, male, female, hermaphrodite, wild, bred for fiber, seeds or medicinal resin – is a vegetable with every dietary essential we can’t synthesize: Essential Amino Acids, Essential Fatty Acids, Essential Cannabinoid acids and hundreds of anti-Cancer compounds.
This is a documentary of how myself and others cured our cancers using an alternative and controversial treatment: cannabis oil. It’s a proven fact that CBD and THC, two of many components in cannabis, shrink tumors and cure cancer. This documentary shows my cancer being cured and explores the history and politics of cannabis and cancer. You will also see samples of many antique cannabis medicine bottles.
Medicinal marijuana oil made from cannabis buds, when ingested thrice daily, for two months, will destroy leukemia and cancer cells. Here is just one study which shows the results: http://www.ncbi.nlm.nih.gov/pubmed/16908594:
Abstract
Plant-derived cannabinoids, including Delta9-tetrahydrocannabinol (THC), induce apoptosis in leukemic cells, although the precise mechanism remains unclear. In the current study, we investigated the effect of THC on the upstream and downstream events that modulate the extracellular signal-regulated kinase (ERK) module of mitogen-activated protein kinase pathways primarily in human Jurkat leukemia T cells. The data showed that THC down-regulated Raf-1/mitogen-activated protein kinase/ERK kinase (MEK)/ERK/RSK pathway leading to translocation of Bad to mitochondria. THC also decreased the phosphorylation of Akt. However, no significant association of Bad translocation with phosphatidylinositol 3-kinase/Akt and protein kinase A signaling pathways was noted when treated cells were examined in relation to phosphorylation status of Bad by Western blot and localization of Bad to mitochondria by confocal analysis. Furthermore, THC treatment decreased the Bad phosphorylation at Ser(112) but failed to alter the level of phospho-Bad on site Ser(136) that has been reported to be associated with phosphatidylinositol 3-kinase/Akt signal pathway. Jurkat cells expressing a constitutively active MEK construct were found to be resistant to THC-mediated apoptosis and failed to exhibit decreased phospho-Bad on Ser(112) as well as Bad translocation to mitochondria. Finally, use of Bad small interfering RNA reduced the expression of Bad in Jurkat cells leading to increased resistance to THC-mediated apoptosis. Together, these data suggested that Raf-1/MEK/ERK/RSK-mediated Bad translocation played a critical role in THC-induced apoptosis in Jurkat cells.
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